3 edition of Neuroendocrine responses to social regulation of puberty in the female house mouse found in the catalog.
Neuroendocrine responses to social regulation of puberty in the female house mouse
|Statement||Kirwin J. Darney, Jr., Jerome M. Goldman, John G. Vandenbergh.|
|Contributions||Goldman, Jerome M., Vandenbergh, John G., United States. Environmental Protection Agency.|
|The Physical Object|
Female puberty was assessed by the opening of the vagina and male puberty by the first copulation plug. The sexes differed in the mean age at puberty, males being older by 13 days in the large, 4 days in the control and 8 days in the small lines. The sexes differed also in Cited by: The Neuroendocrine Regulation of Behavior [The Neuroendocrine Regulation of Behavior THE NEUROENDOCRINE REGULATION OF BEHAVIOR by Schulkin, Jay (Author) on Jan Hardcover] on *FREE* shipping on qualifying offers. The Neuroendocrine Regulation of 4/5(1).
Lucinda Carr is Consultant Paediatric Neurologist at Great Ormond Street Hospital for Children, London, working across the departments of neurology and served as the clinical lead in neurology at the hospital from to Dr Carr's main research interest is in movement disorders and cerebral palsy and she was a member of the team that developed the NICE guidelines on. neuroendocrine regulation can be complete without due consider-ation of the environmental effects of oestrogens on homeostasis. The review by Cruz et al. (5) discusses how neonatal exposure to environmental oestrogens can modify epigenetic programming and produce alterations in reproductive function in adulthood.
Puberty phase involving the maturation of primary sexual characteristics (ovaries in females, testes in males) and secondary sexual characteristics (pubic hair, breast and genital development). This period follows adrenarche by about 2 years and is what most people think of as puberty. The first demonstrable biological change of puberty is the appearance of pulsatile LH release during sleep. As puberty progresses, the frequency and amplitude of LH secretory peaks increase, although peaks are also found during the wake period. At the end of puberty, the difference between sleep and wake LH secretory patterns disappears.
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Neuroendocrine responses to social regulation of puberty in the female house mouse. Darney KJ Jr(1), Goldman JM, Vandenbergh JG. Author information: (1)Department of Zoology, North Carolina State University, Raleigh First estrus is advanced in female house mice exposed to an adult male and delayed in those housed in by: Get this from a library.
Neuroendocrine responses to social regulation of puberty in the female house mouse. [Kirwin J Darney; Jerome M Goldman; John G Vandenbergh; United States.
Environmental Protection Agency.]. Darney KJ, Goldman JM, Vandenbergh JG () Neuroendocrine responses to social regulation of puberty in the female house mouse. Neuroendocrinology – PubMed CrossRef Google Scholar de Roux N, Genin E, Carel JC, Matsuda F, Chaussain JL, Milgrom E () Hypogonadotropic hypogonadism due to loss of function of the KiSS1-derived peptide Cited by: 4.
Introduction. Puberty - the period of becoming first capable of reproducing sexually, marked by maturation of the genital organs, development of secondary sex characteristics, and, in the human and other highly evolved primates 1, by the first occurrence of menstruation in the female  - has not previously provided the central theme of the Geoffrey Harris Memorial by: This chapter discusses the neuroendocrine regulation of puberty.
Puberty is the phase in life when individuals become capable of reproducing, and in the case of females, able to sustain a pregnancy to term.
During puberty, major hormonal, physical, and behavioral changes take place; in humans and other primates, such changes are accompanied by Cited by: Verification of this broad hypothesis will require implementation of the second step proposed for the application of a system biology strategy to the understanding of the neuroendocrine control of puberty, that is, the controlled perturbation of specific genes followed by the global detection of the attendant cellular responses and the Cited by: A book of the names and address of people living in a city.
'Neuroendocrine responses to social regulation of puberty in the female house mouse' -- subject(s): Puberty, Luteinizing hormone. The link between affective and neural responses to social exclusion is reinforced by positive correlations between exclusion-related activation in the ACC/pMFC and self-reported distress (Eisenberger et al., ; Masten et al., ) and negative correlations between orbitofrontal activation and the ‘pain’ of being excluded (Eisenberger et Cited by: 4.
Abstract. The onset of puberty in mammals occurs at a species-typical age and thus is thought to be under genetic control. While heredity plays an important role in establishing the approximate time of puberty (Stone and Barker ), other factors such as nutrition and social stimuli also play an important role in scheduling the time of puberty within the range of ages limited by by: 7.
at the time of rat puberty (26). We observed that the abun-dance of glutamate dehydrogenase, one of the enzymes that catalyzes the synthesis of glutamate (27), increases in the hypothalamus of female rats undergoing puberty. In con-trast, the abundance of glutamine synthase, which catalyzes the metabolism of glutamate into glutamine (27), decreasesFile Size: 1MB.
This chapter discusses the neuroendocrine regulation of puberty. Puberty is the phase in life when individuals become capable of reproducing, and in the case of females, able to sustain a. Kirwin J. Darney has written: 'Neuroendocrine responses to social regulation of puberty in the female house mouse' -- subject(s): Puberty, Luteinizing hormone Asked in Puberty and Adolescence.
Hormonal Regulation of Puberty and Fertility The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government.
The central neuroendocrine regulation of luteinizing hormone-releasing hormone (LHRH) production and gonadotropin secretion initiates the onset of puberty in rats through LH actions on the Leydig.
This research investigates potential neuroendocrine factors controlling and/or affecting the onset of puberty. The overall goal of this research is to use anatomical, physiological, and molecular approaches to learn more about the timely synchronization of events within the hypothalamic-pituitary-ovarian axis that lead to the onset of puberty and to discern how certain toxic substances affect.
The data obtained in the present study indicate that chronic corticosterone exposure throughout adolescent development results in significant and sex-dependent somatic and neuroendocrine changes, and the results also provide an experimental framework for further investigating the impact of corticosterone on metabolic and neuroendocrine function.
Wild animals are animals from Ants to Elephants; represent a natural resource of great significance for most forest-dwelling. Wildlife provides a major part of the animal protein in the diets of rural people in a great many developing countries.
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Neuroendocrine Regulation. and has thusly stretched to examine various interconnections of the endocrine and sensory systems. The neuroendocrine framework is the system by which the hypothalamus keeps up homeostasis, managing proliferation, digestion system, consuming and drinking conduct, vitality usage, osmolarity, and pulse.
A brain-neuroendocrine process occurring primarily in early adolescence that provides stimulation for the rapid physical changes that accompany this period of development puberty Puberty is. Darney KJ Jr Goldman J, Vandenbergh J.
Neuroendocrine responses to social regulation of puberty in the female house mouse. Neuroendocrinology. ;55(4)– [ PubMed ]. Much has been learned concerning the neuroendocrine processes and cellular mechanisms by which steroid hormones influence reproductive behaviors in rodents and other animals.
In this review, a short discussion of hormones and feminine sexual behavior in some rodent species is followed by an outline of the main principles that have been learned from these studies. Examples are given of the. Interestingly, sex differences on the pubertal roles of NKB might exist, as Tac2-/-males did not show overt pubertal alterations (True et al., ), and LH responses to the NKB agonist, senktide, changed variably during postnatal maturation in male and female rats, with persistent responses after puberty being observed only in females (Ruiz Cited by: Enhanced at puberty 1 (EAP1) is a new transcriptional regulator of the female neuroendocrine reproductive axis Sabine Heger,1 Claudio Mastronardi, 2 Gregory A.
Dissen, Alejandro Lomniczi, 2 Ricardo Cabrera, Christian L. Roth,2 Heike Jung, 2 Francesco Cited by: